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奈特醫學圖譜全集(第3版第5卷泌尿系統)(英文版)

  • 作者:(美)弗蘭克·H.奈特//羅山·M.帕特爾//詹姆·蘭德曼|編者:樂偲//閆莉|責編:李元君//白蘭|總主編:馬超
  • 出版社:中國協和醫科大學
  • ISBN:9787567925779
  • 出版日期:2025/08/01
  • 裝幀:平裝
  • 頁數:295
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內容大鋼
    《奈特醫學繪圖圖集》(第3版)(始於1948年,奈特博士與CIBA製藥(諾華製藥前身)合作的單卷本,后拓展為多卷套「綠皮書」)共9卷(14分冊),以簡明扼要、高度直觀的方式,呈現了各器官系統的基礎科學和臨床醫學知識。這套書籍包含精妙絕倫的教學插圖,體現了最新的醫學內容。每卷都是在來自加州大學、賓夕法尼亞大學、梅奧診所、克利夫蘭診所等各領域的教學和醫療專家編輯團隊的指導下進行修訂,將解剖學、生理學、病理學等基礎學科的核心概念,與臨床實踐中常見的疾病問題、醫療診斷和手術治療相融合。本圖集延續了經典的奈特藝術風格,新增並更新了插圖,輔以現代影像圖片,使醫學概念變得生動鮮活——這部不朽之作也由此成為學生、臨床醫生和教育工作者不可或缺的寶貴資源。本冊是《奈特醫學圖譜全集(第3版第5卷泌尿系統)》,內容包括十一個部分。

作者介紹
(美)弗蘭克·H.奈特//羅山·M.帕特爾//詹姆·蘭德曼|編者:樂偲//閆莉|責編:李元君//白蘭|總主編:馬超

目錄
SECTION 1 ANATOMY OF THE URINARY TRACT
  1.1  Kidney: Position and Relations (Anterior View)
  1.2  Kidney: Position and Relations (Posterior Views)
  1.3  Kidney: Position and Relations (Transverse Sections)
  1.4  Kidney: Gross Structure
  1.5  Renal Fascia
  1.6  Kidney: Position, Relations, Gross Structure
  1.7  Bladder: Position, Relations, Gross Structure (Male)
  1.8  Bladder: Position, Relations, Gross Structure (Female)
  1.9  Bladder: Position, Relations, Gross Structure (Coronal Cross Sections)
  1.10  Renal Vasculature: Renal Artery and Vein in Situ
  1.11  Renal Vasculature: Renal Artery Segmental Branches and Intrarenal Arteries
  1.12  Renal Vasculature: Variations in Renal Artery and Vein
  1.14  Innervation of Kidneys and Bladder
  1.15  Innervation of Kidneys and Bladder
  1.16  Innervation Pathways of the Ureter and Bladder
  1.17  Lymphatics of Urinary System
  1.18  Overview of the Nephron
  1.19  Renal Microvasculature
  1.20  Glomerulus: Structure and Histology
  1.21  Glomerulus: Fine Structure
  1.22  Glomerulus: Electron Microscopy
  1.23  Proximal Tubule
  1.24  Thin Limb
  1.25  Distal Tubule
  1.26  Collecting Duct
  1.27  Renal Pelvis, Ureter, and Bladder
SECTION 2 NORMAL AND ABNORMAL DEVELOPMENT
  2.1  Development of Kidney
  2.2  Development of Kidney: Nephron Formation
  2.3  Development of Bladder and Ureter: Formation of Cloaca
  2.4  Development of Bladder and Ureter: Septation
  2.5  Kidney Ascent
  2.6  Renal Ascent and Ectopia: Normal Kidney Ascent and Pelvic Kidney
  2.7  Renal Rotation and Malrotation
  2.8  Anomalies in Number of Kidneys: Bilateral Renal Agenesis
  2.9  Anomalies in Number of Kidneys: Unilateral Renal Agenesis
  2.10  Anomalies in Number of Kidneys: Supernumerary Kidney
  2.11  Renal Fusion
  2.12  Renal Dysplasia
  2.13  Simple Cysts
  2.14  Kidney Masses
  2.15  Simple Cysts: Bosniak Classification of Cyst
  2.16  Polycystic Kidney Disease: Gross Appearance
  2.17  Polycystic Kidney Disease: Radiographic Findings
  2.18  Medullary Sponge Kidney
  2.19  Nephronophthisis/Medullary Cystic Kidney Disease Complex
  2.20  Ectopic Ureter: Radiographic Findings and Laparoscopic Repair
  2.21  Vesicoureteral Reflux: Normal Development of the Inferior Vena Cava
  2.22  Vesicoureteral Reflux: Mechanism and Grading

  2.23  Vesicoureteral Reflux: Voiding Cystourethrograms
  2.24  Ureteral Duplication: Complete
  2.25  Ureteral Duplication: Incomplete
  2.26  Ectopic Ureter
  2.27  Ureterocele: Gross and Fine Appearance
  2.28  Ureterocele: Radiographic Findings
  2.29  Prune Belly Syndrome: Appearance of Abdominal Wall
  2.30  Prune Belly Syndrome: Appearance of Kidneys
  2.31  Epispadias Exstrophy Complex
  2.32  Epispadias Exstrophy Complex: Bladder Exstrophy
  2.33  Bladder Duplication and Septation
  2.34  Anomalies of the Urachus
  2.35  Posterior Urethral Valves: Congenital Bladder Outlet Obstruction
  2.36  Posterior Urethral Valves: Radiographic Findings
SECTION 3 PHYSIOLOGY
  3.1  Basic Functions and Homeostasis
  3.2  Clearance and Renal Plasma Flow
  3.3  Glomerular Filtration Rate
  3.4  Glomerular Filtration Rate: Calculation
  3.5  Secretion and Reabsorption: Tubular Reabsorption and Saturation Kinetics
  3.6  Secretion and Reabsorption: Fractional Excretion (Clearance Ratios)
  3.7  Renal Handling of Sodium and Chloride: Nephron Sites of Sodium Reabsorption
  3.8  Renal Handling of Sodium and Chloride: Response to Extracellular Fluid Contraction
  3.9  Renal Handling of Sodium and Chloride: Response to Extracellular Fluid Expansion
  3.10  Renal Handling of Potassium
  3.11  Renal Handling of Calcium, Phosphate, and Magnesium
  3.12  Countercurrent Multiplication: Model of the Countercurrent Multiplier
  3.13  Countercurrent Multiplication: Models to Demonstrate Principle of Countercurrent Exchange and Maintenance of the Medullary Osmotic Gradient
  3.14  Urine Concentration and Dilution: Long-Looped Nephron (ADH Present)
  3.15  Urine Concentration and Dilution: Long-Looped Nephron (ADH Absent)
  3.16  Antidiuretic Hormone
  3.17  Renin-Angiotensin-Aldosterone System
  3.18  Tubuloglomerular Feedback and Renin-Angiotensin-Aldosterone System
  3.19  Acid-Base Balance: Roles of Chemical Buffers, Lungs, and Kidneys in Acid-Base Handling
  3.20  Acid-Base Balance: Renal Bicarbonate Reabsorption
  3.21  Acid-Base Balance: Renal Bicarbonate Synthesis and Proton Excretion
  3.22  Acid-Base Balance: Acidosis and Alkalosis
  3.23  Additional Functions: Erythropoiesis and Vitamin D
  3.24  Renal Tubular Acidosis: Proximal
  3.25  Renal Tubular Acidosis: Classic Distal
  3.26  Renal Tubular Acidosis: Inipidus
  3.27  Nephrogenic Diabetes Inipidus: Major Causes and Symptoms
SECTION 4 KIDNEY DISEASES
  4.1  Overview of Acute Kidney Injury: Causes
  4.2  Overview of Acute Kidney Injury: Possible Urine Sediment Findings
  4.3  Acute Tubular Necrosis: Causes, Pathophysiology, and Histopathologic Findings
  4.4  Acute Tubular Necrosis: Histopathologic Findings
  4.5  Overview of Nephrotic Syndrome: Pathophysiology
  4.6  Overview of Nephrotic Syndrome: Causes
  4.7  Overview of Nephrotic Syndrome: Presentation and Diagnosis

  4.8  Minimal Change Disease: Causes and Presentation
  4.9  Minimal Change Disease: Histopathologic Findings
  4.10  Focal Segmental Glomerulosclerosis: Causes, Clinical Features, and Histopathologic Findings
  4.11  Focal Segmental Glomerulosclerosis: Histopathologic Findings (Continued)
  4.12  Membranous Nephropathy: Causes and Clinical Features
  4.13  Membranous Nephropathy: Histopathologic Findings
  4.14  Overview of Glomerulonephritis: Clinical Features and Histopathologic Findings
  4.15  Overview of Glomerulonephritis: Histopathologic Findings
  4.16  Immunoglobulin A Nephropathy: Causes and Clinical Features
  4.17  Immunoglobulin A Nephropathy: Histopathologic Findings
  4.18  Immunoglobulin A Nephropathy: Histopathologic Findings (Continued)
  4.19  Postinfectious Glomerulonephritis: Causes and Clinical Features
  4.20  Postinfectious Glomerulonephritis: Histopathologic Findings
  4.21  Postinfectious Glomerulonephritis: Histopathologic Findings (Continued)
  4.22  Membranoproliferative Glomerulonephritis: Causes, Features, and Assessment
  4.23  Membranoproliferative Glomerulonephritis: Classical Pathway of Complement Activation
  4.24  Membranoproliferative Glomerulonephritis: Histopathologic Findings
  4.25  Rapidly Progressive Glomerulonephritis
  4.26  Hereditary Nephritis (Alport Syndrome)/ Thin Basement Membrane Nephropathy: Pathophysiology and Clinical Features
  4.27  Hereditary Nephritis (Alport Syndrome)/Thin Basement Membrane Nephropathy: Electron Microscopy Findings
  4.28  Hereditary Nephritis: Causes and Clinical Features
  4.29  Acute Interstitial Nephritis: Histopathologic Findings
  4.30  Chronic Tubulointerstitial Nephritis and Analgesic Nephropathy
  4.31  Chronic Tubulointerstitial Nephritis: Histopathologic Findings
  4.32  Thrombotic Microangiopathy: General Features
  4.33  Thrombotic Microangiopathy: Hemolytic Uremic Syndrome
  4.34  Thrombotic Microangiopathy: Thrombotic Thrombocytopenic Purpura
  4.35  Renal Vein Thrombosis
  4.36  Renal Artery Stenosis: Pathophysiology of Renovascular Hypertension
  4.37  Renal Artery Stenosis: Causes
  4.38  Congestive Heart Failure: Types of Left Heart Failure and Effects on Renal Function
  4.39  Congestive Heart Failure: Effects of Left Heart Failure on Renal Blood Flow and Tubular Function
  4.40  Hepatorenal Syndrome: Proposed Pathophysiology
  4.41  Hepatorenal Syndrome: Symptoms and Diagnosis
  4.42  Chronic and Malignant Hypertension: Major Causes
  4.43  Chronic and Malignant Hypertension: Renal Histopathology (Chronic)
  4.44  Chronic and Malignant Hypertension: Renal Histopathology (Malignant)
  4.45  Diabetic Nephropathy: Diabetes Mellitus
  4.46  Diabetic Nephropathy
  4.47  Amyloidosis: Deposition Sites and Histopathology
  4.48  Amyloidosis: Histopathologic Findings
  4.49  Lupus Nephritis: Diagnostic Criteria
  4.50  Lupus Nephritis: Renal Histopathology (Class I and II Lesions)
  4.51  Lupus Nephritis: Renal Histopathology (Class III and IV Les
  4.58  Preeclampsia: Clinical Definition and Potential Mechanism of Pathogenesis
  4.59  Preeclampsia/Nephrotic Syndrome
  4.60  Preeclampsia: HELLP Syndrome and Edampsia
  4.61  Immunoglobulin A Vasculitis: Diagnostic Criteria
  4.62  Immunoglobulin A Vasculitis: Additional Clinical Features
  4.63  Fabry Disease
  4.64  Cystinosis: Pathophysiology and Renal Fanconi Syndrome
  4.65  Cystinosis: Extrarenal Manifestations
  4.66  Overview of Chronic Kidney Disease: Staging System and Major Causes
  4.67  Overview of Chronic Kidney Disease: Normal and Abnormal GFR
  4.68  Overview of Chronic Kidney Disease: Calcium and Phosphate Metabolism
  4.69  Overview of Chronic Kidney Disease: Mechanism of Progression and Complications
  4.70  Overview of Chronic Kidney Disease: Uremia
……
SECTION 5 URINARY TRACT INFECTIONS
SECTION 6 URINARY TRACT OBSTRUCTIONS
SECTION 7 TRAUMATIC INJURIES
SECTION 9 NEOPLASMS
SECTION 10 THERAPEUTICS
SECTION 11 PROSTATE
Selected References
Index

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